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Monast (Finasteride)

Monast is used for the treatment of male pattern hair loss (also known as androgenetic alopecia) in men aged 18-41. Male pattern hair loss is a common condition thought to be caused by a combination of genetic factors and a particular hormone, called dihydrotestosterone (DHT). DHT contributes to shortening of the growth phase of the hair and to thinning of the hair. In the scalp, Monast specifically lowers the levels of DHT by blocking an enzyme (Type II 5-alpha reductase) that converts testosterone to DHT. Only men with mild to moderate, but not complete hair loss can expect to benefit from the use of Monast. In most of the men treated with Monast for 5 years, the progression of hair loss was slowed, and at least half of these men also had some kind of improved hair growth.

Other names for this medication:
Finasteride, Androfin, Avertex, Daric, Fenered, Finahair, Finalo, Finalop, Finar, Finara, Finast, Finax, Fincar, Finpecia, Firide, Monast, Prohair, Propecia, Proscalpin, Proscar, Prosteride, Reduprost, Renacidin, Sutrico, Symasteride, Tealep, Tensen,

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Trizol, Vonum, Lasix, Kenazol, Nansen, Vivanza, Nabratin, Difloxin, Levozine, Ezetrol, Trizole, Difene, Betamox, Acyvir, Rantec, Rosasol, Aldep, Larotid, Vostar, Cooltime, Lancid, Naxy, Relaxol, Pandon, Dezor, Cleocin, Spirolang, Lostat, Glipizide, Medacin, Anipryl, Isoproterenol, Ropinirol, Deroxat, Contiflo, Lanacine, Amoval, Actalipid, Incivek, Spiron, Artroxicam, Atorlip, Planor,

Monast comes as a tablet to take by mouth. It is usually taken once a day with or without food. Take Monast at around the same time every day. Follow the directions on your prescription label carefully, and ask your doctor or pharmacist to explain any part you do not understand. Take Monast exactly as directed. Do not take more or less of it or take it more often than prescribed by your doctor.

If you are taking Monast to treat BPH, you should know that Monast may control your condition, but will not cure it. It may take at least 6 months before your symptoms improve. Continue to take Monast even if you feel well. Do not stop taking Monast without talking to your doctor.

If you are taking Monast to treat male pattern hair loss, it may take at least 3 months before you see any improvement because hair loss and growth happen slowly over time. However, you should expect to see improvement during the first 12 months of your treatment. If you have taken Monast for 12 months and have not noticed any improvement, further treatment probably will not help. Talk to your doctor about whether you should continue your treatment.

Monast will only slow hair loss while you are taking the medication. Continue to take Monast even if you have already noticed an improvement. Do not stop taking Monast without talking to your doctor. You will probably lose any hair that grew back while you were taking Monast during the first 12 months after you stop taking the medication..

Ask your pharmacist or doctor for a copy of the manufacturer's information for the patient.

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Creatinine clearance and urea clearance in peritoneal dialysis: what to do in case of discrepancy. Economic implications of newborn screening for cystic fibrosis: a cost of illness retrospective cohort study. Participants were volunteers functioning in the cognitively normal range. Childhood sleep problems: association with prenatal factors and maternal distress/depression.

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The genes for the variable domain of the Fab were cloned and sequenced and the primary amino acid sequence was deduced from this information. Optimal drug and other treatment (this should be optimal treatment available in that country). Search for inhibitors against herpes simplex virus type-I in cell extracts derived from human lymphoblastoid cell lines.

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We prospectively assessed the incidence of bleeding complications after microdiscectomy in patients treated with a single 30 mg intraoperative dose of Ketorolac subsequent to wound closure. Influence of advanced recipient and donor age on the outcome of corneal transplantation. Death, dying and grief in a transcultural context: application of the Giger and Davidhizar assessment model. The clinical and morphologic features of fatal cases suggest that these represented the extreme of a biologic continuum, with collapse of defense mechanisms despite chemotherapy. Polysomnographic recordings, which included hippocampal field potentials at the hippocampal fissure, were carried out in rats for 8 h during the light phase of the circadian cycle.

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The CARD10 (CARMA3)-Bcl10-Malt1 signalosome functions as a link between G protein-coupled receptor (GPCR) signaling and proinflammatory NF-kappaB activation. This result provides an animal model for understanding the underlying mechanisms of the development of SMA and will allow efficient selection against the disease in cattle.

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Regulation by Src homology 2 domain-containing protein tyrosine phosphatase substrate-1 of alpha-galactosylceramide-induced antimetastatic activity and Th1 and Th2 responses of NKT cells. Because of the efficient screening mammography programs, most of the breast cancers diagnosed today in the United States and European Union are in early stage and are treated with limited surgery.

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These observation suggest that the irreversibility of experimental CCl4 cirrhosis of the liver is associated with a disturbance in the mechanisms of collagen degradation, which may be a deficiency in collagenase activity, a change in the susceptibility of the substrate, or a combination of both factors. We found substantial variations in thyroid hormone levels and clinical presentation, but most individuals were without symptoms of thyroid disorder.

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These atypical-appearing aneurysms may erroneously be considered to be thrombosed or be mistaken for other common lesions such as small tumors and be dismissed as nonvascular. Experimental congenital infection with cytomegalovirus: a guinea pig model. Acute adrenal cortex insufficiency as cause of shock and sudden death in unexpected immersion, severe phenomenon of endocrine system nonadaptability to the stress of trauma and cold Both functions of the fusion molecule, fibrin-specific binding and plasminogen activation, were fully preserved.

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Practice guidelines and professional opinion regarding disclosure appear to depart from the actual experience reported by clinicians, patients, and family members. It is suggested that rotational flexibility combined with other deformities could be evaluated in future studies on prediction of pulmonary function from the measurements of the deformity. However, there is no functional evidence to support such a hypothesis. Chondroitin 4-sulfate proteoglycans constitute the predominant ones produced by activated human monocytes/macrophages.

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The results of the present study suggest that experimental diabetes is associated with changes of the adenylate cyclase and/or phosphodiesterase enzyme activities in skeletal muscle resulting in an increased responsiveness to adrenaline. pylori infection was detected by either a positive rapid urease test or the presence of the bacteria on histology.

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Alkaline phosphatase activity in the developing teeth of the rat. This finding strongly supports the conclusion that both the liganded and the unliganded forms of the binding protein interact with the membrane components of the transport system. The B cell immunoglobulin repertoire of an immunized rabbit was immortalized by the combinatorial cloning of the rearranged variable domains of light (VL) and heavy (VH) chains. The recommendations are relatively straightforward and have been widely promoted both in guidelines and reviews.

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Spearman rank correlations measured associations among glioma grade, rCBVmax, and histogram measures. We have demonstrated the feasibility of preparing caged peptides by derivatizing a single amino acid side chain in peptides up to 20 amino acids long. Unlike all previously reported cases of primary hepatic neuroendocrine tumors, which have been endocrinologically asymptomatic, the patient had a Zollinger-Ellison syndrome apparently cured by surgical resection of the tumor. Repair of a traumatic scleral rupture with scleral imbrication and BioGlue. Seventeen nonpregnant, healthy women with a history of gestational diabetes and 20 nonpregnant control women of similar age without previous diabetes.

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There is an urgent need to renew UK policies of harm reduction in order to support perceptions that HCV is avoidable and preventable. Qualitative aspects of hydrogen-deuterium exchange in the 1H, 13C, and 15N nuclear magnetic resonance spectra of viomycin in aqueous solution.

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New consecutive high-dose chemotherapy modality with fractionated blood stem cell support in the treatment of high-risk pediatric solid tumors: a feasibility study. It occurs most frequently and appears to be most refractory in patients with IBM. Induced binding was completely inhibited by the phosphatidylinositol 3-kinase (PI 3-K) inhibitor wortmannin.

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The seroprevalence of toxoplasmosis in sheep, goats and pigs in Zimbabwe. Four patients have been treated from April to November 1994, three female and one male, ranging in age from 13 to 31 years. Medium chains of adaptor complexes AP-1 and AP-2 recognize leucine-based sorting signals from the invariant chain.

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